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Angiotensin II Receptor Blocker/Prescription

AZILSARTAN MEDOXOMIL

AZILSARTAN MEDOXOMIL

Clinical safety rating

caution

Comprehensive clinical and safety monograph for AZILSARTAN MEDOXOMIL (AZILSARTAN MEDOXOMIL).


Mechanism of Action

Angiotensin II receptor blocker (ARB) that selectively inhibits angiotensin II binding to AT1 receptors, reducing vasoconstriction, aldosterone secretion, and sympathetic activity.

What the body does with it

MetabolismPrimarily metabolized by CYP2C9 to inactive metabolites; also undergoes esterase-mediated hydrolysis to azilsartan.
ExcretionBiliary/fecal (55% unchanged), renal (42% as inactive metabolites, <1% unchanged)
Half-lifeTerminal half-life approximately 11 hours; supports once-daily dosing with sustained antihypertensive effect over 24 hours.
Protein bindingHigh (>99%) to serum albumin.
Volume of DistributionVd of about 16 L (0.23 L/kg for a 70 kg individual); indicates limited extravascular distribution.
BioavailabilityOral bioavailability approximately 60% under fed conditions (food reduces absorption); absolute bioavailability not determined in humans.
Onset of ActionOral: 1-2 hours for peak plasma concentration; antihypertensive effect begins within 2-3 hours.
Duration of ActionDuration of antihypertensive effect exceeds 24 hours; steady state achieved by day 7.
Molecular Weight456.49

Classification & Brands

Dosing & administration

40 mg orally once daily. May increase to 80 mg once daily if needed.

Dosage formTABLET
Renal impairmentNo dose adjustment required for GFR ≥15 mL/min/1.73 m². Not recommended for GFR <15 mL/min/1.73 m² due to lack of data.
Liver impairmentNo dose adjustment required for mild to moderate hepatic impairment (Child-Pugh A and B). Not recommended for severe hepatic impairment (Child-Pugh C) due to lack of data.
Pediatric useNot approved for use in pediatric patients (safety and efficacy not established).
Geriatric useNo specific dose adjustment recommended; initiate at 40 mg once daily. Monitor renal function and blood pressure carefully due to increased sensitivity.

Use during pregnancy

1st trimesterAvoid. Teratogenic risk from fetal exposure to angiotensin II receptor antagonists during the first trimester is low but cannot be excluded; alternatives preferred.
2nd trimesterContraindicated. Associated with fetal renal dysfunction, oligohydramnios, skull ossification defects, and neonatal anuria/hypotension; risk is highest in second and third trimesters.
3rd trimesterContraindicated. Same risks as second trimester; may cause fetal injury or death.

Clinical note

Comprehensive clinical and safety monograph for AZILSARTAN MEDOXOMIL (AZILSARTAN MEDOXOMIL).

Placental transferCrosses placenta in animals; human data limited but presumed to cross due to low molecular weight and known fetal toxicity.
BreastfeedingExcretion into human milk is unknown; due to potential for adverse effects on infant kidney function, use during breastfeeding is not recommended. If used, monitor infant for hypotension and renal impairment.
Lactation RatingL4 - Possibly Hazardous
Teratogenic RiskFirst trimester: Limited human data; animal studies show no teratogenicity. Second and third trimesters: Drugs acting directly on the renin-angiotensin system can cause fetal oligohydramnios, fetal renal dysfunction, skull ossification defects, and neonatal anuria, hypotension, and death.
Fetal MonitoringMonitor fetal growth via ultrasound, assess amniotic fluid volume (oligohydramnios), and fetal renal function. Postnatal monitoring of infant for hypotension and hyperkalemia.
Fertility EffectsNo human data; animal studies at high doses showed reduced fertility. Effects likely mediated by altered renin-angiotensin system.

Warnings & precautions

■ FDA Black Box Warning

none

Side Effect Profile

Serious Effects

Absolute Contraindications

Hypersensitivity to azilsartan medoxomil or any componentHistory of angioedema related to previous ARB therapyConcomitant use with aliskiren in patients with diabetes mellitusPregnancy (second and third trimesters)

Clinical Precautions

PrecautionsFetal toxicity: avoid use in pregnancy, Hypotension in volume-depleted patients, Renal impairment: monitor renal function, Hyperkalemia: monitor potassium levels
Food/DietaryNo significant food interactions; can be taken with or without food. Avoid excessive potassium intake from high-potassium foods (e.g., bananas, oranges, spinach, potatoes) or potassium-containing salt substitutes. Limit alcohol intake as it may increase blood pressure or cause dizziness.

Clinical Tips & Counseling

Clinical PearlsAzilsartan medoxomil has the highest affinity for AT1 receptors among ARBs; may cause a rapid decrease in blood pressure in volume-depleted patients; avoid use in pregnancy (Category D); monitor renal function and serum potassium; less CYP450 interaction potential than losartan or irbesartan; can be taken without regard to meals; dose adjustment not required in mild-to-moderate hepatic impairment.
Patient AdviceTake once daily at the same time each day with or without food. · Avoid becoming dehydrated; drink adequate fluids unless directed otherwise. · Do not use if pregnant or planning to become pregnant; notify your doctor immediately if pregnancy occurs. · Do not take with aliskiren if you have diabetes or renal impairment. · Report any signs of angioedema (swelling of face, lips, tongue, difficulty breathing) or severe dizziness. · May cause dizziness, especially during first few days; avoid driving until you know how the medication affects you. · Avoid potassium supplements and salt substitutes containing potassium unless approved by your doctor. · Do not stop taking the medication without talking to your doctor.

AZILSARTAN MEDOXOMIL Interactions

Loading safety data…

This overview is compiled from peer-reviewed clinical sources and FDA labeling. It's here to support — not replace — clinical judgment. Always verify dosing against your institution's current protocols before prescribing.

On this page

Mechanism of ActionDosing & administrationUse during pregnancyWarnings & precautionsDrug interactions

Compare with

ATACANDATACAND HCTBENICARBYVALSONEDARBI

External sources

DailyMed (NIH) PubMed OpenFDA