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Registry Hub
Peer-Reviewed Evidence
HomeDrug RegistryCompareATMEKSI vs DUTASTERIDE
Comparative Pharmacology

ATMEKSI vs DUTASTERIDE Comparison

Head-to-head clinical analysis & difference comparison: details on mechanism of action, dosing, half-life, interactions, and maternal-fetal safety.

Clinical EssentialsPharmacokineticsSpecial PopulationsSafety & MonitoringPregnancy & LactationClinical Insights
Differential Analysis

ATMEKSI vs DUTASTERIDE

Clinician-reviewed, head-to-head comparison of mechanism, dosing, pharmacokinetics, and safety profiles.

View ATMEKSI Monograph View DUTASTERIDE Monograph
ATMEKSI
PDE5 Inhibitor
Category C
DUTASTERIDE
5-alpha Reductase Inhibitor
Category D/X
TL;DR — Key Differences
  • Drug class: ATMEKSI is a PDE5 Inhibitor; DUTASTERIDE is a 5-alpha Reductase Inhibitor.
  • Half-life: ATMEKSI has a half-life of Terminal elimination half-life is 12 hours; renally impaired patients have prolonged half-life up to 24 hours.; DUTASTERIDE has Terminal half-life approximately 3-4 weeks (21-35 days) in young adults; 5-6 weeks in elderly; supports once-daily dosing due to slow elimination..
  • No direct drug-drug interaction has been documented between ATMEKSI and DUTASTERIDE.
  • Pregnancy: ATMEKSI is rated Category C; DUTASTERIDE is rated Category D/X.

Last clinically reviewed: July 2026 · OpiCalc Medical Review Team

Clinical Essentials

ATMEKSI
DUTASTERIDE
Mechanism of Action
ATMEKSI

ATMEKSI (atazanavir/cobicistat) is a fixed-dose combination of atazanavir, an HIV-1 protease inhibitor that inhibits viral protease, preventing cleavage of viral polyproteins and resulting in immature non-infectious virions, and cobicistat, a pharmacokinetic enhancer that inhibits CYP3A, increasing atazanavir exposure.

DUTASTERIDE

Competitive inhibitor of type II and type I 5α-reductase isoenzymes, blocking conversion of testosterone to dihydrotestosterone (DHT) in prostate, hair follicles, and other tissues.

Indications
ATMEKSI

Treatment of HIV-1 infection in adults and pediatric patients weighing at least 35 kg,Off-label use as part of antiretroviral therapy in treatment-experienced patients with viral suppression

DUTASTERIDE

FDA: Treatment of symptomatic benign prostatic hyperplasia (BPH) in men with an enlarged prostate,FDA: Reduce risk of acute urinary retention,FDA: Reduce need for BPH-related surgery,Off-label: Male androgenetic alopecia

Standard Dosing
ATMEKSI

1.5 mg/kg IV every 4 weeks

DUTASTERIDE

0.5 mg orally once daily.

Direct Interaction
ATMEKSI
No Direct Interaction
DUTASTERIDE
No Direct Interaction

Pharmacokinetics

ATMEKSI
DUTASTERIDE
Half-Life
ATMEKSI

Terminal elimination half-life is 12 hours; renally impaired patients have prolonged half-life up to 24 hours.

DUTASTERIDE

Terminal half-life approximately 3-4 weeks (21-35 days) in young adults; 5-6 weeks in elderly; supports once-daily dosing due to slow elimination.

Metabolism
ATMEKSI

Atazanavir is metabolized by CYP3A4; cobicistat is metabolized by CYP3A and to a minor extent by CYP2D6.

DUTASTERIDE

Extensively metabolized in liver via CYP3A4 and CYP1A2; minor metabolism by CYP2C8, CYP2C9, CYP2C19, and CYP2D6.

Excretion
ATMEKSI

Primarily renal (80% unchanged) and biliary/fecal (15% as metabolites).

DUTASTERIDE

Primarily fecal (70%) as metabolites; renal excretion accounts for <5% unchanged drug.

Protein Binding
ATMEKSI

95% bound to albumin and alpha-1-acid glycoprotein.

DUTASTERIDE

>99% bound to albumin and alpha-1 acid glycoprotein; high affinity.

VD (L/kg)
ATMEKSI

2.0 L/kg, indicating extensive tissue distribution.

DUTASTERIDE

Approximately 300-500 L (3-5 L/kg), indicating extensive tissue distribution, particularly to prostate and seminal vesicles.

Bioavailability
ATMEKSI

Oral: 60-70% due to first-pass metabolism.

DUTASTERIDE

Oral: Approximately 60% (range 40-80%) with food; not administered parenterally.

Special Populations

ATMEKSI
DUTASTERIDE
Renal Adjustments
ATMEKSI

GFR 15-29 m L/min: 1.0 mg/kg IV every 4 weeks; GFR <15 m L/min: not recommended

DUTASTERIDE

No dose adjustment required for renal impairment (including dialysis).

Hepatic Adjustments
ATMEKSI

Child-Pugh A: no adjustment; Child-Pugh B: 1.0 mg/kg IV every 4 weeks; Child-Pugh C: not recommended

DUTASTERIDE

Contraindicated in Child-Pugh Class C; use with caution in mild to moderate impairment (Child-Pugh A/B) with no specific dose adjustment established.

Pediatric Dosing
ATMEKSI

Age 2-17 years: 1.5 mg/kg IV every 4 weeks; maximum 120 mg per dose

DUTASTERIDE

Not indicated in pediatric patients; safety and efficacy not established.

Geriatric Dosing
ATMEKSI

No specific adjustment; monitor renal function and reduce dose if GFR <30 m L/min

DUTASTERIDE

No specific dose adjustment required; monitor for adverse effects (e.g., dizziness, orthostatic hypotension) due to age-related comorbidities.

Safety & Monitoring

ATMEKSI
DUTASTERIDE
Black Box Warnings
ATMEKSI
FDA Black Box Warning

None

DUTASTERIDE
FDA Black Box Warning

No FDA black box warning.

Warnings/Precautions
ATMEKSI

Hepatotoxicity, especially in patients with pre-existing liver disease or elevated transaminases,Nephrolithiasis and cholelithiasis,Cardiac conduction abnormalities (PR interval prolongation),Risk of developing resistance if not used with other antiretrovirals,Renal impairment (cobicistat decreases estimated creatinine clearance)

DUTASTERIDE

Risk of high-grade prostate cancer in men aged 50-79 with elevated PSA and previous negative biopsy (see PLCO trial),Increased risk of sexual adverse events (impotence, decreased libido, ejaculation disorders) that may persist after discontinuation,Elevated PSA levels: use caution when interpreting PSA values; establish new baseline after 6 months of treatment

Contraindications
ATMEKSI

Concomitant use with drugs highly dependent on CYP3A for clearance (e.g., alfuzosin, rifampin, ergot derivatives, St. John's wort, lovastatin, simvastatin, sildenafil for pulmonary arterial hypertension),Severe hepatic impairment (Child-Pugh Class B or C)

DUTASTERIDE

Women of childbearing potential (pregnancy category X; risk of fetal harm due to inhibition of 5α-reductase),History of hypersensitivity to dutasteride or other 5α-reductase inhibitors,Pediatric patients

Adverse Reactions
ATMEKSI
Data Pending
DUTASTERIDE
Data Pending
Food Interactions
ATMEKSI

Avoid alcohol (may exacerbate CNS effects). Grapefruit juice may increase atomoxetine exposure; limit consumption. High-fat meals do not significantly affect absorption.

DUTASTERIDE

No clinically significant food interactions. May be taken with or without food. Grapefruit juice does not affect dutasteride levels to a clinically relevant extent.

Pregnancy & Lactation

ATMEKSI
DUTASTERIDE
Teratogenic Risk
ATMEKSI

First trimester: Limited human data; animal studies show no teratogenicity at therapeutic doses. Second and third trimesters: No known fetal risks. Avoid use during organogenesis unless benefit outweighs risk.

DUTASTERIDE

Dutasteride is contraindicated in pregnancy. It is a 5α-reductase inhibitor that can inhibit the conversion of testosterone to dihydrotestosterone (DHT), potentially causing abnormal development of external genitalia in male fetuses. Risk extends throughout all trimesters due to potential disruption of androgen-mediated development in male fetuses during the first trimester and cumulative effects from drug accumulation in adipose tissue. No adequate human studies exist; animal studies show teratogenicity in male offspring at clinically relevant doses.

Lactation Summary
ATMEKSI

Not recommended during breastfeeding. M/P ratio unknown. Excreted in animal milk; potential for serious adverse reactions in nursing infants.

DUTASTERIDE

No data on dutasteride in human milk. M/P ratio unknown. Dutasteride is highly lipophilic and likely excreted in breast milk. Because of potential adverse effects on the nursing infant (e.g., interference with androgen-mediated development in male infants), breastfeeding is contraindicated during therapy and for at least 6 months after the last dose due to long half-life (approximately 5 weeks).

Pregnancy Dosing
ATMEKSI

No dose adjustment required in pregnancy. Pharmacokinetic profile unchanged.

DUTASTERIDE

No dose adjustment studies in pregnancy because dutasteride is contraindicated. Pharmacokinetic changes in pregnancy (increased volume of distribution, altered metabolism) could affect dutasteride levels, but no adjustments are recommended as drug should not be used. If inadvertently used, discontinue immediately and monitor for adverse effects.

Maternal Safety Status
ATMEKSI
Category C
DUTASTERIDE
Category D/X

Clinical Insights

ATMEKSI
DUTASTERIDE
Clinical Pearls
ATMEKSI

ATMEKSI (atomoxetine) is a selective norepinephrine reuptake inhibitor (NRI) indicated for ADHD. It has a slower onset of action (2-4 weeks) compared to stimulants. Monitor for hepatotoxicity and suicidal ideation, especially in children and adolescents. Use cautiously with hepatic impairment (reduce dose) and CYP2D6 poor metabolizers (need lower dose). Avoid concurrent MAOIs. May cause orthostatic hypotension and urinary retention.

DUTASTERIDE

Monitor PSA levels cautiously, as dutasteride reduces serum PSA by approximately 50% after 6 months; double the PSA value for comparison to untreated men. Do not handle crushed or broken capsules if pregnant or planning pregnancy, as absorption through skin may cause fetal harm. Assess for signs of high-grade prostate cancer before initiating therapy, as dutasteride may increase the risk of Gleason 8-10 tumors. Onset of symptom relief may take 3-6 months; do not discontinue prematurely. Avoid concomitant use with strong CYP3A4 inhibitors (e.g., ritonavir, ketoconazole) as they increase dutasteride exposure.

Patient Counseling
ATMEKSI

Take ATMEKSI exactly as prescribed; do not change dose without consulting your doctor.,It may take 2-4 weeks to notice improvement in symptoms.,Avoid alcohol and grapefruit juice as they may affect drug levels.,Report any signs of liver problems (yellowing of skin/eyes, dark urine, abdominal pain) or suicidal thoughts immediately.,May cause dizziness or fainting, especially when standing up; rise slowly.,Do not stop abruptly without medical advice.

DUTASTERIDE

Take exactly as prescribed; do not stop or change dose without consulting your doctor.,Swallow the capsule whole; do not chew or open it.,It may take 3 to 6 months to see improvement in symptoms.,Avoid handling leaking or crushed capsules if you are a woman who is or may become pregnant; wash area immediately with soap and water if skin contact occurs.,Do not donate blood for at least 6 months after your last dose to prevent exposure to a pregnant woman.,Report any breast lumps, pain, or nipple discharge promptly.,You will need regular blood tests for PSA level monitoring; inform your doctor that you are taking dutasteride.,Dutasteride can decrease sperm count and may affect fertility; discuss this with your doctor if planning to father a child.

Safety Verification

Known Interactions

ATMEKSI Risks

No interactions on record

DUTASTERIDE Risks3
Dutasteride + Sulfisoxazole
moderate

"Dutasteride, a 5α-reductase inhibitor, may inhibit cytochrome P450 enzymes, particularly CYP3A4, which is involved in the metabolism of sulfisoxazole. This inhibition can lead to decreased clearance of sulfisoxazole, resulting in elevated plasma concentrations. Increased sulfisoxazole levels may potentiate its adverse effects, including hypersensitivity reactions, crystalluria, and hematologic toxicity such as agranulocytosis."

Dutasteride + Nelfinavir
moderate

"Concomitant use of dutasteride, a 5α-reductase inhibitor, with nelfinavir, a protease inhibitor and potent CYP3A4 inhibitor, is predicted to increase the serum concentration of nelfinavir. This occurs because dutasteride may inhibit the metabolism of nelfinavir via competition for CYP3A4, leading to elevated nelfinavir levels and an increased risk of adverse effects such as gastrointestinal disturbances, hepatotoxicity, and metabolic complications. Clinical monitoring for toxicity and dose adjustments are warranted."

Dutasteride + Itraconazole
moderate

"Dutasteride, a 5α-reductase inhibitor, is metabolized primarily by CYP3A4 and to a lesser extent by CYP2D6. Itraconazole is a potent inhibitor of CYP3A4 and also inhibits P-glycoprotein. Coadministration leads to significantly increased serum concentrations of dutasteride, raising the risk of adverse effects such as gynecomastia, sexual dysfunction, and depression. The effect on itraconazole levels is minimal and clinically irrelevant."

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Clinical Q&A

Frequently Asked Questions

Common clinical questions about ATMEKSI vs DUTASTERIDE, answered by our medical review team.

1. What is the main difference between ATMEKSI and DUTASTERIDE?

ATMEKSI is a PDE5 Inhibitor that works by ATMEKSI (atazanavir/cobicistat) is a fixed-dose combination of atazanavir, an HIV-1 protease inhibitor that inhibits viral protease, preventing cleavage of viral polyproteins and resulting in immature non-infectious virions, and cobicistat, a pharmacokinetic enhancer that inhibits CYP3A, increasing atazanavir exposure.. DUTASTERIDE is a 5-alpha Reductase Inhibitor that works by Competitive inhibitor of type II and type I 5α-reductase isoenzymes, blocking conversion of testosterone to dihydrotestosterone (DHT) in prostate, hair follicles, and other tissues.. They differ in pharmacokinetic profiles, FDA-approved indications, and side effect profiles.

2. Which is stronger: ATMEKSI or DUTASTERIDE?

Potency comparisons between ATMEKSI and DUTASTERIDE depend on the specific clinical indication. These are agents from distinct pharmacological classes and are not directly interchangeable by dose. A physician or clinical pharmacist should guide any therapeutic switching decisions.

3. What is the standard dosing for ATMEKSI vs DUTASTERIDE?

The standard adult dose of ATMEKSI is: 1.5 mg/kg IV every 4 weeks. The standard adult dose of DUTASTERIDE is: 0.5 mg orally once daily.. Dosing should always be individualized based on indication, renal and hepatic function, age, and other patient factors.

4. Can you take ATMEKSI and DUTASTERIDE together?

No direct drug-drug interaction has been formally documented between ATMEKSI and DUTASTERIDE in current clinical databases. However, individual patient risk factors including other medications, organ function, and comorbidities should always be evaluated by a qualified healthcare provider.

5. Are ATMEKSI and DUTASTERIDE safe during pregnancy?

The maternal-fetal safety profiles differ. ATMEKSI is classified as Category C. First trimester: Limited human data; animal studies show no teratogenicity at therapeutic doses. Second and third trimesters: No known fetal risks. Avoid use during organogenesis u. DUTASTERIDE is classified as Category D/X. Dutasteride is contraindicated in pregnancy. It is a 5α-reductase inhibitor that can inhibit the conversion of testosterone to dihydrotestosterone (DHT), potentially causing abnorm. Always consult a maternal-fetal medicine specialist before taking either drug during pregnancy or lactation.