Logo

OpiCalc

FavoritesSpecialtiesDrugsGuidelinesMost Used

Quick Access

Favorites
Most Used

All Specialties

OpiCalc Logo
Clinical CalculatorsDrugsGuidelines
SpecsDrugsGuides
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
‌
OpiCalc Logo

OpiCalc

Easy, fast, and private medical tools for clinicians. Always free.

No Login Required
Ready for the Bedside

Resources

About UsEditorial PolicyMedical DisclaimerPrivacy PolicyTerms of UseCookie Policy

Support

Contact Us

Clinical Notice:OpiCalc is not a substitute for professional clinical judgment. Always verify dosages and guidelines.

OpiCalc © 2018-2026

•

All Rights Reserved

Registry Hub
Peer-Reviewed Evidence
HomeDrug RegistryCompareCARDENE vs ADALAT
Comparative Pharmacology

CARDENE vs ADALAT Comparison

Head-to-head clinical analysis & difference comparison: details on mechanism of action, dosing, half-life, interactions, and maternal-fetal safety.

Clinical EssentialsPharmacokineticsSpecial PopulationsSafety & MonitoringPregnancy & LactationClinical Insights
Differential Analysis

CARDENE vs ADALAT

Clinician-reviewed, head-to-head comparison of mechanism, dosing, pharmacokinetics, and safety profiles.

View CARDENE Monograph View ADALAT Monograph
CARDENE
Calcium Channel Blocker
Category C
ADALAT
Calcium Channel Blocker
Category C
TL;DR — Key Differences
  • Half-life: CARDENE has a half-life of 1.5-2 hours (terminal); prolonged in hepatic impairment (up to 6-8 hours); ADALAT has Terminal elimination half-life: 2-5 hours (immediate-release); 8-14 hours (extended-release). Context: shorter half-life necessitates multiple daily dosing for immediate-release; extended-release allows once-daily dosing..
  • No direct drug-drug interaction has been documented between CARDENE and ADALAT.
  • Pregnancy: CARDENE is rated Category C; ADALAT is rated Category C.

Last clinically reviewed: July 2026 · OpiCalc Medical Review Team

Clinical Essentials

CARDENE
ADALAT
Mechanism of Action
CARDENE

Cardene (nicardipine) is a dihydropyridine calcium channel blocker that inhibits the transmembrane influx of calcium ions into vascular smooth muscle and cardiac muscle. It dilates peripheral arterioles, reducing systemic vascular resistance and blood pressure, and also has coronary vasodilatory effects.

ADALAT

Dihydropyridine calcium channel blocker; inhibits calcium ion influx across cardiac and vascular smooth muscle cells, reducing peripheral vascular resistance and blood pressure.

Indications
CARDENE

Hypertension (chronic stable angina is not an approved indication in the US; however, it is used off-label for short-term treatment of hypertension in hospitalized patients and for subarachnoid hemorrhage to prevent vasospasm.,Off-label: short-term management of hypertension in hospitalized patients, prevention of cerebral vasospasm after subarachnoid hemorrhage.

ADALAT

Hypertension,Chronic stable angina,Vasospastic angina (Prinzmetal's angina)

Standard Dosing
CARDENE

20-40 mg orally three times daily.

ADALAT

10-20 mg orally three times daily; extended-release: 30-60 mg orally once daily; maximum 120 mg/day.

Direct Interaction
CARDENE
No Direct Interaction
ADALAT
No Direct Interaction

Pharmacokinetics

CARDENE
ADALAT
Half-Life
CARDENE

1.5-2 hours (terminal); prolonged in hepatic impairment (up to 6-8 hours)

ADALAT

Terminal elimination half-life: 2-5 hours (immediate-release); 8-14 hours (extended-release). Context: shorter half-life necessitates multiple daily dosing for immediate-release; extended-release allows once-daily dosing.

Metabolism
CARDENE

Hepatic metabolism primarily via cytochrome P450 isoenzymes CYP3A4 and CYP2C8, with minor contributions from CYP2D6.

ADALAT

Hepatic via CYP3A4; extensive first-pass metabolism; metabolites are inactive.

Excretion
CARDENE

Renal: 60% as metabolites, 10% unchanged; Fecal: 35%

ADALAT

Renal: 70-80% as metabolites; Fecal: 15-20% as metabolites; <1% unchanged in urine

Protein Binding
CARDENE

>95% bound to albumin and alpha-1 acid glycoprotein

ADALAT

92-98% bound to plasma proteins (albumin and alpha-1-acid glycoprotein)

VD (L/kg)
CARDENE

0.6-1.2 L/kg (large Vd due to extensive tissue binding)

ADALAT

0.8-1.2 L/kg. Clinical meaning: indicates extensive tissue distribution, consistent with high lipophilicity.

Bioavailability
CARDENE

Oral: 35-60% (first-pass metabolism; increased with hepatic disease)

ADALAT

Oral immediate-release: 45-60% (due to first-pass metabolism); extended-release: 60-85% (due to slower release and reduced first-pass effect).

Special Populations

CARDENE
ADALAT
Renal Adjustments
CARDENE

For GFR < 30 m L/min: initiate at 20 mg orally twice daily.

ADALAT

No dose adjustment required for GFR ≥30 m L/min; for GFR <30 m L/min, use with caution and reduce initial dose by 50%.

Hepatic Adjustments
CARDENE

Child-Pugh class B or C: reduce dose by 50% and titrate slowly.

ADALAT

Child-Pugh Class A: no adjustment; Class B: reduce dose by 50%; Class C: avoid use or reduce by 75%.

Pediatric Dosing
CARDENE

Not established; safety and efficacy not determined.

ADALAT

0.25-0.5 mg/kg/dose orally every 6-8 hours; maximum 3 mg/kg/day. Extended-release not recommended.

Geriatric Dosing
CARDENE

Initiate at 20 mg orally twice daily; titrate cautiously.

ADALAT

Start at 10 mg orally twice daily; titrate slowly due to increased sensitivity and risk of hypotension.

Safety & Monitoring

CARDENE
ADALAT
Black Box Warnings
CARDENE
FDA Black Box Warning

No FDA black box warning.

ADALAT
FDA Black Box Warning

None

Warnings/Precautions
CARDENE

Hypotension (especially in patients with ventricular dysfunction or those receiving beta-blockers), increased angina (rare, more common with dihydropyridines), peripheral edema, congestive heart failure (use caution in patients with severe left ventricular dysfunction), hepatic impairment (reduce dose), renal impairment (monitor blood pressure), abrupt discontinuation may cause angina exacerbation.

ADALAT

May cause hypotension, especially in patients on beta-blockers or with poor cardiac reserve,Risk of increased angina and/or myocardial infarction upon initiation or dose increase,Peripheral edema,Stevens-Johnson syndrome and toxic epidermal necrolysis (rare),Hepatic impairment,Exacerbation of angina on withdrawal

Contraindications
CARDENE

Hypersensitivity to nicardipine or any dihydropyridine, advanced aortic stenosis (may reduce coronary perfusion), second- or third-degree AV block (unless paced), sick sinus syndrome (unless paced), cardiogenic shock, concomitant use with strong CYP3A4 inducers (e.g., rifampin) due to decreased efficacy.

ADALAT

Hypersensitivity to nifedipine,Cardiogenic shock,Significant aortic stenosis,Concurrent use with rifampin,Pregnancy (category C)

Adverse Reactions
CARDENE
Data Pending
ADALAT
Data Pending
Food Interactions
CARDENE

Grapefruit and grapefruit juice increase nicardipine exposure and should be avoided. High-fat meals may reduce absorption; take consistently with or without food. Alcohol may enhance hypotensive effects.

ADALAT

Avoid grapefruit and grapefruit juice; they inhibit CYP3A4 and increase nifedipine serum concentrations, leading to enhanced hypotensive effects and risk of toxicity. Grapefruit interaction persists for 24 hours; separate consumption by at least 4 hours if unavoidable, but preferable to avoid entirely. Avoid alcohol which can increase hypotension. High-fat meals may reduce absorption of extended-release formulations; take consistently with or without food.

Pregnancy & Lactation

CARDENE
ADALAT
Teratogenic Risk
CARDENE

Cardene (nicardipine) is classified as FDA Pregnancy Category C. In animal studies, nicardipine was associated with embryotoxicity, fetotoxicity, and teratogenicity at high doses. There are no adequate and well-controlled studies in pregnant women. Use in the first trimester should be avoided if possible; in the second and third trimesters, potential benefits must outweigh risks, particularly due to possible maternal hypotension and fetal hypoxia.

ADALAT

First trimester: Limited human data; animal studies show embryotoxicity. Second/third trimester: May cause fetal hypoxia due to maternal hypotension; risk of preterm labor inhibition. Category C.

Lactation Summary
CARDENE

Nicardipine is excreted in human breast milk. The M/P ratio is approximately 0.7. Limited data suggest that concentrations in breast milk are low relative to therapeutic doses, but the effects on the nursing infant are unknown. Caution is advised.

ADALAT

Excreted in breast milk; M/P ratio ~0.85. Consider risks versus benefits; monitor infant for hypotension.

Pregnancy Dosing
CARDENE

Pregnancy may alter the pharmacokinetics of nicardipine due to increased plasma volume and altered hepatic metabolism. However, specific dose adjustment recommendations are not established. In hypertensive emergencies, intravenous nicardipine is often used at standard doses with careful titration to avoid maternal hypotension. Oral doses may require titration based on response, starting at 20 mg three times daily.

ADALAT

No standard dose adjustment; monitor clinical response and blood pressure; may require lower doses due to vasodilation effects.

Maternal Safety Status
CARDENE
Category C
ADALAT
Category C

Clinical Insights

CARDENE
ADALAT
Clinical Pearls
CARDENE

CARDENE (nicardipine) is a dihydropyridine calcium channel blocker used intravenously for acute hypertension. Onset is rapid (1-2 minutes), making it ideal for hypertensive emergencies. It is metabolized hepatically; reduce dose in hepatic impairment. Does not cause reflex tachycardia as prominently as other dihydropyridines. Contraindicated in advanced aortic stenosis. Can be used for hypertension during pregnancy but with caution.

ADALAT

Adalat (nifedipine) is a dihydropyridine calcium channel blocker. Use immediate-release capsules only for hypertensive emergencies, not chronic treatment due to risk of reflex tachycardia and unpredictable hypotension. Extended-release formulations are preferred for stable angina and hypertension. Avoid grapefruit juice as it increases nifedipine levels via CYP3A4 inhibition. Monitor for peripheral edema, gingival hyperplasia, and constipation. Contraindicated in cardiogenic shock, severe aortic stenosis, and within 4 weeks of myocardial infarction.

Patient Counseling
CARDENE

Take exactly as prescribed; do not stop abruptly.,May cause dizziness or lightheadedness; avoid driving until you know how it affects you.,Avoid grapefruit and grapefruit juice as they may increase drug levels.,Report irregular heartbeat, shortness of breath, or swelling of the ankles or feet.,Keep all appointments for blood pressure monitoring.

ADALAT

Swallow extended-release tablets whole; do not crush, chew, or split.,Avoid grapefruit and grapefruit juice while taking this medication.,Report persistent swelling of ankles/feet, gum tenderness or bleeding, or severe dizziness.,Do not stop abruptly; taper under medical supervision to avoid rebound hypertension.,Take at the same time each day; if a dose is missed, skip it if near next dose.,May cause dizziness; avoid driving until you know how it affects you.,Increase fluid and fiber intake to prevent constipation.,Store at room temperature away from light and moisture.

Safety Verification

Known Interactions

CARDENE Risks

No interactions on record

ADALAT Risks

No interactions on record

Compare Alternatives

Related Drug Comparisons

Explore head-to-head clinical comparisons of other medications in the same therapeutic classes.

CARDENE vs ADALAT CCCalcium Channel Blocker
ADALAT vs ADALAT CCCalcium Channel Blocker
CARDENE vs AFEDITAB CRCalcium Channel Blocker
ADALAT vs AFEDITAB CRCalcium Channel Blocker
CARDENE vs AMVAZCalcium Channel Blocker
ADALAT vs AMVAZCalcium Channel Blocker
CARDENE vs CADUETCalcium Channel Blocker + HMG-CoA Reductase Inhibitor
ADALAT vs CADUETCalcium Channel Blocker + HMG-CoA Reductase Inhibitor
CARDENE vs CALANCalcium Channel Blocker
Clinical Q&A

Frequently Asked Questions

Common clinical questions about CARDENE vs ADALAT, answered by our medical review team.

1. What is the main difference between CARDENE and ADALAT?

CARDENE is a Calcium Channel Blocker that works by Cardene (nicardipine) is a dihydropyridine calcium channel blocker that inhibits the transmembrane influx of calcium ions into vascular smooth muscle and cardiac muscle. It dilates peripheral arterioles, reducing systemic vascular resistance and blood pressure, and also has coronary vasodilatory effects.. ADALAT is a Calcium Channel Blocker that works by Dihydropyridine calcium channel blocker; inhibits calcium ion influx across cardiac and vascular smooth muscle cells, reducing peripheral vascular resistance and blood pressure.. They differ in pharmacokinetic profiles, FDA-approved indications, and side effect profiles.

2. Which is stronger: CARDENE or ADALAT?

Potency comparisons between CARDENE and ADALAT depend on the specific clinical indication. These are both Calcium Channel Blocker agents and are not directly interchangeable by dose. A physician or clinical pharmacist should guide any therapeutic switching decisions.

3. What is the standard dosing for CARDENE vs ADALAT?

The standard adult dose of CARDENE is: 20-40 mg orally three times daily.. The standard adult dose of ADALAT is: 10-20 mg orally three times daily; extended-release: 30-60 mg orally once daily; maximum 120 mg/day.. Dosing should always be individualized based on indication, renal and hepatic function, age, and other patient factors.

4. Can you take CARDENE and ADALAT together?

No direct drug-drug interaction has been formally documented between CARDENE and ADALAT in current clinical databases. However, individual patient risk factors including other medications, organ function, and comorbidities should always be evaluated by a qualified healthcare provider.

5. Are CARDENE and ADALAT safe during pregnancy?

The maternal-fetal safety profiles differ. CARDENE is classified as Category C. Cardene (nicardipine) is classified as FDA Pregnancy Category C. In animal studies, nicardipine was associated with embryotoxicity, fetotoxicity, and teratogenicity at high doses. . ADALAT is classified as Category C. First trimester: Limited human data; animal studies show embryotoxicity. Second/third trimester: May cause fetal hypoxia due to maternal hypotension; risk of preterm labor inhibiti. Always consult a maternal-fetal medicine specialist before taking either drug during pregnancy or lactation.