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Chelating Agent/Prescription

EDETATE CALCIUM DISODIUM

EDETATE CALCIUM DISODIUM

Clinical safety rating

caution

Comprehensive clinical and safety monograph for EDETATE CALCIUM DISODIUM (EDETATE CALCIUM DISODIUM).


Mechanism of Action

Chelates heavy metals (e.g., lead, cadmium) by forming stable complexes with divalent and trivalent cations, which are then excreted in urine.

What the body does with it

MetabolismNot metabolized; excreted unchanged in urine, primarily via glomerular filtration.
ExcretionPrimarily renal (90-100% as chelated lead complex within 24-48 hours); minimal biliary/fecal excretion (<5%).
Half-lifeTerminal elimination half-life approximately 1.5-3 hours for the intact chelate; prolonged to 20-40 hours in lead-intoxicated patients due to redistribution of lead from bone.
Protein bindingNegligible (<5%); primarily binds lead in extracellular fluid.
Volume of Distribution0.3-0.5 L/kg (confined mainly to extracellular space; does not penetrate cells or CNS).
BioavailabilityIV or IM: 100% (not absorbed orally).
Onset of ActionIV: Immediate chelation of lead in plasma; IM: 30-60 minutes; no oral absorption.
Duration of ActionDuration of chelation effect is 6-12 hours per dose; repeated dosing required for sustained lead mobilization.
Molecular Weight374.27

Classification & Brands

Dosing & administration

Edetate calcium disodium is administered intravenously or intramuscularly. For lead poisoning: 1000 mg/m²/day IV continuous infusion or in divided doses every 12 hours; alternatively 50 mg/kg/day IV or IM in divided doses every 8-12 hours. Maximum 3000 mg/day. Duration typically 5 days, repeat after 2 days rest. For other heavy metal toxicity: 50 mg/kg/day IV or IM in divided doses every 8-12 hours for 3-5 days.

Dosage formINJECTABLE
Renal impairmentGFR > 50 mL/min: no adjustment. GFR 30-50 mL/min: reduce dose by 50%. GFR 15-29 mL/min: reduce dose by 75%. GFR < 15 mL/min or dialysis: contraindicated or use with extreme caution at 10-20% of normal dose with monitoring.
Liver impairmentNo specific guidelines for Child-Pugh based modifications. Use with caution in severe hepatic impairment due to potential for electrolyte disturbances and nephrotoxicity.
Pediatric useFor lead poisoning: 1000 mg/m²/day IV continuous infusion or in divided doses every 12 hours; or 50 mg/kg/day IV or IM in divided doses every 8-12 hours. Maximum 3000 mg/day. Course: 5 days, may repeat after 2 days rest. For other indications: weight-based dosing similar to adults (50 mg/kg/day).
Geriatric useStart at low end of dosing range (e.g., 30-40 mg/kg/day) due to age-related decreased renal function. Monitor renal function and electrolytes closely. Avoid use in patients with pre-existing renal impairment without dose reduction.

Use during pregnancy

1st trimesterLimited human data; animal studies not available. Avoid use unless clearly needed.
2nd trimesterUse only if clearly needed; potential for zinc depletion in mother and fetus.
3rd trimesterUse only if clearly needed; risk of zinc depletion and maternal/fetal toxicity.

Clinical note

Comprehensive clinical and safety monograph for EDETATE CALCIUM DISODIUM (EDETATE CALCIUM DISODIUM).

Placental transferCrosses placenta in animals; unknown in humans.
BreastfeedingExcretion in milk unknown; may cause zinc deficiency in infant due to chelation. Avoid or use caution.
Lactation RatingL3 (Moderately Safe)
Teratogenic RiskFDA Pregnancy Category C. In animal studies, edetate calcium disodium has been shown to be teratogenic (skeletal anomalies) at doses approximately 0.25 times the human dose. There are no adequate and well-controlled studies in pregnant women. The drug should be used during pregnancy only if the potential benefit justifies the potential risk to the fetus. First trimester: Risk cannot be ruled out; potential for teratogenic effects. Second and third trimesters: Potential fetal toxicity from chelation of essential minerals; avoid use unless necessary.
Fetal MonitoringMonitor serum calcium, magnesium, and zinc levels during therapy due to chelation of essential divalent cations. Monitor renal function (BUN, serum creatinine, urinalysis) before and during treatment because the drug is nephrotoxic. Obtain baseline and periodic ECG due to potential hypocalcemia-induced QT prolongation. Monitor for signs of tetany or seizures in both mother and fetus. Assess fetal growth and well-being with ultrasound if prolonged therapy is required.
Fertility EffectsNo specific human studies on fertility. Animal studies have not been reported. However, chelation of essential metals like zinc may potentially affect spermatogenesis and oocyte maturation. Chronic use may impair fertility due to depletion of trace elements.

Warnings & precautions

■ FDA Black Box Warning

Black box warning for nephrotoxicity, particularly at high doses; may cause fatal renal failure. Avoid in patients with renal disease unless benefits outweigh risks. Monitor renal function closely during therapy.

Side Effect Profile

Serious Effects

Absolute Contraindications

Hypersensitivity to edetate calcium disodiumSevere renal impairment (anuria or oliguria)Wilson's disease (due to copper depletion)Hepatolenticular degeneration

Clinical Precautions

PrecautionsNephrotoxicity - monitor BUN, creatinine, and urinalysis at baseline and during therapy., Neurotoxicity - may cause tremors, myoclonus, and seizures, especially with high doses., Hepatotoxicity - monitor liver function tests., Arrhythmias - may cause QT prolongation; monitor ECG., Hypocalcemia - monitor serum calcium levels; may cause tetany., Use with caution in patients with asthma or allergic history., Not recommended for prophylaxis of heavy metal poisoning.
Food/DietaryNo specific food restrictions, but maintain adequate hydration. Avoid alcohol due to potential renal effects.

Clinical Tips & Counseling

Clinical PearlsEdetate calcium disodium is used for chelation therapy in heavy metal poisoning, particularly lead. Monitor renal function closely due to risk of nephrotoxicity. Administer via slow IV infusion (over 8-12 hours) after dilution to minimize irritation. Contraindicated in anuria or severe renal impairment. Rapid infusion may cause hypocalcemic tetany; ensure adequate hydration to enhance lead excretion. Use with caution in patients with arrhythmias due to potential electrolyte disturbances.
Patient AdviceThis medication is used to remove lead from your body. · You will receive this drug as an intravenous infusion over several hours. · Drink plenty of fluids during treatment to help flush out lead. · Report any muscle cramps, numbness, or tingling immediately. · Your kidney function and electrolyte levels will be monitored during treatment. · Avoid taking other medications without consulting your doctor, especially those affecting the kidneys.

EDETATE CALCIUM DISODIUM Interactions

Loading safety data…

This overview is compiled from peer-reviewed clinical sources and FDA labeling. It's here to support — not replace — clinical judgment. Always verify dosing against your institution's current protocols before prescribing.

On this page

Mechanism of ActionDosing & administrationUse during pregnancyWarnings & precautionsDrug interactions

Compare with

BAFIERTAMBALCALCIUM DISODIUM VERSENATECHEMETCUPRIMINE

External sources

DailyMed (NIH) PubMed OpenFDA