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Registry Hub
Antiarrhythmic (Class Ia)/Discontinued

PROCAINAMIDE HCL

PROCAINAMIDE HCL

Clinical safety rating

safe

Other drugs that prolong the QT interval increase risk of torsades de pointes Can cause lupus-like syndrome and blood dyscrasias.


What is PROCAINAMIDE HCL?

Other drugs that prolong the QT interval increase risk of torsades de pointes Can cause lupus-like syndrome and blood dyscrasias.

Indications & Uses

Treatment of documented ventricular arrhythmias (e.g., sustained ventricular tachycardia) that are life-threateningOff-label: Atrial fibrillation, atrial flutter, supraventricular tachycardia, Wolff-Parkinson-White syndrome

Side Effects

Lupus-like syndrome

Compare PROCAINAMIDE HCL vs CIN-QUIN →View all Antiarrhythmic (Class Ia) drugs →

Mechanism of Action

Class Ia antiarrhythmic agent; blocks sodium channels, decreases phase 0 slope of action potential, prolongs refractory period, and increases action potential duration.

What the body does with it

MetabolismHepatic via N-acetyltransferase (NAT2) to N-acetylprocainamide (NAPA); also undergoes hydrolysis to p-aminobenzoic acid.
ExcretionPrimarily renal (50-60% unchanged via glomerular filtration and tubular secretion) with 10-30% as N-acetylprocainamide (NAPA) metabolite; minor biliary/fecal (<5%).
Half-lifeTerminal elimination half-life: 2.5-4.7 hours (3 hours typical) in normal renal function; prolonged to 11-20 hours in renal impairment; NAPA half-life 6-8 hours (prolonged in renal failure).
Protein binding15-25% bound primarily to alpha-1-acid glycoprotein and albumin.
Volume of Distribution1.5-2.5 L/kg (approximates total body water; indicates extensive tissue distribution).
BioavailabilityOral: 75-95% (immediate-release); IM: 100%; IV: 100%.
Onset of ActionIV: 1-5 minutes; IM: 10-15 minutes; Oral: 30-60 minutes (immediate-release).
Duration of ActionIV/IM: 3-4 hours (antiarrhythmic effect); Oral immediate-release: 3-6 hours; extended-release: 8-12 hours (dosing interval).
Molecular Weight235.32

Classification & Brands

Dosing & administration

For life-threatening ventricular arrhythmias, IV: Loading dose: 100 mg administered at a rate of 25-50 mg/min, may repeat every 5 minutes until arrhythmia suppressed or up to a total of 500-1000 mg. Maintenance: IV infusion 1-4 mg/min. Oral: 250-500 mg every 3-6 hours; maximum 4 g/day.

Dosage formInjectable
Renal impairmentCrCl >50 mL/min: No adjustment. CrCl 10-50 mL/min: Administer every 6-12 hours. CrCl <10 mL/min: Administer every 12-24 hours.
Liver impairmentChild-Pugh Class A: No adjustment. Child-Pugh Class B: Reduce dose by 25-50%. Child-Pugh Class C: Reduce dose by 50-75% or avoid use.
Pediatric useIV: Loading dose: 15 mg/kg over 30-60 minutes, followed by maintenance infusion of 20-80 mcg/kg/min; maximum 2 g/day. Oral: 15-50 mg/kg/day in divided doses every 3-6 hours; maximum 4 g/day.
Geriatric useStart at lower end of dosing range due to age-related decreased renal function and increased risk of hypotension and arrhythmias. Monitor renal function and adjust accordingly.

Use during pregnancy

1st trimesterProcainamide crosses the placenta. Limited human data; potential risk of neonatal lupus and heart block. Use only if clearly needed.
2nd trimesterSimilar considerations as t1. Monitor fetal heart rate for bradycardia.
3rd trimesterMay cause maternal hypotension and reduce uterine blood flow. Risk of neonatal bradycardia and QT prolongation. Avoid near term.

Clinical note

Other drugs that prolong the QT interval increase risk of torsades de pointes Can cause lupus-like syndrome and blood dyscrasias.

FDA categoryAnimal
Placental transferCrosses placenta extensively; achieves fetal serum concentrations approximately 60-70% of maternal levels.
BreastfeedingProcainamide and its active metabolite NAPA are excreted into breast milk in low concentrations. Monitor infant for bradycardia, hypotension, and GI effects. Generally considered compatible with breastfeeding.
Lactation RatingL2 (Safer)
Teratogenic RiskFDA pregnancy category C. Procainamide crosses the placenta. In first trimester, risk of congenital anomalies is not well-studied; animal studies show no teratogenicity but use only if clearly needed. In second and third trimesters, chronic use may be associated with fetal bradycardia, QT prolongation, and neonatal lupus syndrome (transient). Avoid in pregnancy if possible.
Fetal MonitoringMonitor maternal ECG (QTc interval), procainamide and NAPA serum concentrations, renal function, complete blood count (risk of agranulocytosis). Fetal monitoring: assess fetal heart rate and rhythm, consider fetal echocardiography for QT interval if maternal treatment prolonged.
Fertility EffectsNo known adverse effects on human fertility. Animal studies have not been conducted. Procainamide does not affect gonadotropin levels. Limited data; no specific fertility impairment reported.

Warnings & precautions

■ FDA Black Box Warning

Procanamide can cause agranulocytosis, leukopenia, and thrombocytopenia; fatal blood dyscrasias have occurred. Frequent blood counts are recommended.

Side Effect Profile

Common EffectsLupus-like syndrome
Serious Effects

Absolute Contraindications

Complete heart blockSecond-degree AV block (unless paced)Systemic lupus erythematosusKnown hypersensitivity to procainamide or ester-type anestheticsTorsade de pointesConcurrent use with other drugs that prolong QT interval

Clinical Precautions

PrecautionsMay cause lupus erythematosus-like syndrome (especially with slow acetylator phenotype); proarrhythmic effects (torsades de pointes); hypotension; bone marrow suppression; hepatotoxicity; potentiation of neuromuscular blocking agents.
Food/DietaryNo significant food interactions reported. However, changes in dietary salt intake may affect blood pressure control; avoid excessive caffeine or alcohol as they may trigger arrhythmias. Maintain consistent potassium and magnesium intake; severe electrolyte disbalances can alter drug effect.

Clinical Tips & Counseling

Clinical PearlsProcainamide HCL is a Class IA antiarrhythmic used for atrial and ventricular arrhythmias. It can cause lupus-like syndrome, especially in slow acetylators; monitor ANA titers. Renal dose adjustment is critical due to active metabolite N-acetylprocainamide (NAPA) accumulation. Watch for QT prolongation and torsades de pointes; avoid with other QT-prolonging drugs. Administer IV slowly to avoid hypotension; oral loading requires hepatic first-pass consideration.
Patient AdviceTake this medication exactly as prescribed; do not double doses if missed. · Report any symptoms of lupus (joint pain, rash, fever, chest pain) immediately. · Avoid sudden position changes to prevent dizziness from low blood pressure. · Tell your doctor all other medications, especially other heart rhythm drugs. · You may need regular blood tests to monitor drug levels and organ function. · Do not stop taking this medicine abruptly; sudden stop may worsen arrhythmia. · If using extended-release tablets, do not crush or chew them.

PROCAINAMIDE HCL Interactions

Loading safety data…

This overview is compiled from peer-reviewed clinical sources and FDA labeling. It's here to support — not replace — clinical judgment. Always verify dosing against your institution's current protocols before prescribing.

On this page

Mechanism of ActionDosing & administrationUse during pregnancyWarnings & precautionsDrug interactions

Compare with

CIN-QUINDEXTROMETHORPHAN HYDROBROMIDE AND QUINIDINE SULFATEDISOPYRAMIDE PHOSPHATENORPACENORPACE CR

External sources

DailyMed (NIH) PubMed OpenFDA